Glenohumeral Dislocation

Glenohumeral Joint dislocations account for almost half of all dislocation incidents, occurring when the Head of the Humerus is displaced from the Glenoid Fossa of Scapula (SOURCE-2). Dislocation of the joint represents the most severe form of Glenohumeral Instability that is usually the result of a single traumatic event (SOURCE-11). Through a shared mechanism Glenoid Labrum Tear s, Hill-Sachs Lesions and other structural compromise often occurs concomitantly.


Pathomechanics

The inherently unstable structure of the Glenohumeral Joint leaves it dependent on external stabilisers (detailed on Glenohumeral Instability ) which may be compromised during a traumatic event. The precise mechanism/ direction of injury implicated certain tissues and likelyhood of particular concomitant injuries.

Traumatic Unidirectional Dislocation

Traumatic Anterior Dislocations account for the vast majority (~85-98%) of initial Glenohumeral Dislocations (SOURCE-12). This form of tramatic injury is most often derived from indirect mechanisms where the arm is suddenly loaded in a variable position of combined GH Joint - Abduction , GH Joint - External Rotation and GH Joint - Extension (SOURCE-12+13). Common examples of this position include activites that reflect the Apprehension Test such as overhead throwing or a posterior FOOSH (SOURCE-12+13). Given the tramatic nature of this injury, anterior dislocations often occur (~80%) concomitantly with Hill-Sachs Lesions (SOURCE-13). While most Hill-Sachs Lesions effect less that 30% of the proximal Humerus ’ articular surface, those that exceed this number are expected to play a notable role in reccurence (SOURCE-46). The sequelae that follows a traumatic anterior dislocation typically includes (SOURCE-13+14):

Forming a positive feedback-loop, these symptoms predispose further events of instability.

Posterior Dislocations

Although considerably less represented, the second most common form of Glenohumeral Dislocation is the Posterior Dislocation, accounting for ~2-5% of all shoulder dislocations (SOURCE-14+15). Similarly, these dislocations are often attributed to high velocity impacts (such as motor vehicle accidents), strong muscle contractions (such as a Seizure) or direct trauma to the anterior Humeral Head (SOURCE-14+15). The mechanism behind posterior dislocations typically involves forceful GH Joint - Adduction with GH Joint - Internal Rotation and there is associations with Reverse Hill-Sachs Lesions (SOURCE-14+15). Posterior Dislocations are often (~50-79%) misdiagnosed at initial presentation (SOURCE-14+15).

Inferior Dislocations

Inferior Dislocations, also known asLuxatio Erecta, are the least common form of Glenohumeral Dislocation , accounting for 0.5-1% (SOURCE-16+10). This injury is most often attributed to Hyperabduction from a significant GH Joint - Abduction force which causes the Neck of the Humerus to straighten against the Acromion to compromise the inferior Glenohumeral Joint Capsule and cause inferior migration of the Humeral Head (SOURCE-16+10). Another less reported mechanism involves axial loading of the abducted arm (SOURCE-10). In either instance the vast majority (~80%) of Inferior Dislocations are accompanied by concomitant injury to the following structures (SOURCE-16+10):

The most common long-term complication to follow inferior dislocations is Adhesive Capsulitis (SOURCE-10).

Recurrence

The recurrence of Glenohumeral Instability events is a significant concern as it is both common and associated with a sequelae. Recurrence rates following acute-traumatic Glenohumeral Dislocation were found to be as high as 47% following non-operative management and most often occurred within a year of the initial event (SOURCE-17). For those who underwent surgery, recurrence rates may still be as high as 26% (SOURCE-18). Recurrence is roughly 3 times more likely in males and 13x more likely in those under the age of 20 (SOURCE-17). Recurrent instability is associated with an increased risk of degeneration of the Glenoid, Humeral Head and Glenohumeral Joint Capsule and the number of events was correlated to the extent of damage (SOURCE-17). Bone loss is present in 70-90% of recurrent instability cases (SOURCE-18). Similar degenerative changes were seen in the Synovial Membrane and Collagen structure of those with a history of traumatic instability, which included a denuded Synovial Membrane and Subsynovial Edema (58% of cases), increased vascularity (83%) and increased cellularity (25%) (SOURCE-19).

Bone Loss

Glenoid or Humeral Head Bone loss is common following recurrent episodes of Dislocation, particularly in adolescents (SOURCE-9). This phenomenon further perpetuates the risk of recurrence in a proportional manner where the greater the bone loss the greater the risk (SOURCE-9).

Prevalence

The prevalence of anterior and posterior dislocations is estimated to be 96% and 2-4%, respectively (SOURCE-2). This incidence increases for males aged 21-30 and females 61-80 (SOURCE-2).


Pathology

The following list contains pathologies that often relate to glenohumeral dislocation, as either predisposing factors, concomitant conditions, or sequelae:

Glenoid Labrum Tear - anterior tears predominantly presented with Instability, with Pain as the main a symptom in less than a quarter of instances (SOURCE-20). Conversely, for posterior tears Pain was the primary complaint in the majority of cases, with instability only accounting for 21% (SOURCE-20). Repeated dislocations can lead to detachment of the Glenoid Labrum from the Fossa and/ or damage to the Glenohumeral Capsular Ligaments (SOURCE-3)

  • Bankart Lesion - following a traumatic episode 97% of patients with anterior instability were found to have a Bankart Lesion (SOURCE-21+4)

  • SLAP Lesion - third most common soft-tissue lesion that occurs following traumatic anterior dislocation, associated with roughly a quarter (~23%) (SOURCE-22)

  • Humeral Avulsion of the Glenohumeral Ligaments - estimated to be associated with 7.5-9.3% of primary dislocations and a larger proportion of initial dislocations in those over the age of 35 (SOURCE-23)

Hill-Sachs Lesion - Impression Fractures of the Head of the Humerus are associated with 65-71% of Dislocations and 100% of recurrent Glenohumeral Instability (SOURCE-6).

Scapular Dyskinesis - as displayed in Scapulohumeral Rhythm , the Scapulothoracic Joint and Glenohumeral Joint share the complex but coordinated task of appropriately positioning the Glenoid Fossa to maximise stability of The Shoulder Girdle in a highly mobile environment. Insufficiency from one joint appears to be compensated for by the other, however this appears to come at the expense of movement quality and may compromise associated soft-tissues. Glenohumeral Instability often results in reduced Scapulothoracic Joint - Upward Rotation and a corresponding increase in Scapulothoracic Joint - Protraction , particularly if instability is multidirectional (SOURCE-9). Asymmetric Scapulothoracic motion is also disproportionately higher in those with anteroinferior instability (SOURCE-13).

Rotator Cuff Tear - shares a similar bidirectional relationship with Glenohumeral Joint instability as other shoulder pathologies. Rotator Cuff weakness is associated with anterior instability and recurrence of unstable events (SOURCE-24). Conversely, instability may alter the length-tension relationship of these muscles and predispose injury. Rotator Cuff Tears often occur concomitantly with Dislocations (SOURCE-25).

Nerve Lesions

The majority of nerve lesions following a Glenohumeral Dislocation are benign (SOURCE-7):


Assessment

Observation

Range of Motion

The direction(s) of instability will determine which Range of Motions are either Pain ful or lax/vulnerable. Posterior Dislocations for example, limit or ruin GH Joint - Abduction and GH Joint - External Rotation while these same ranges become excessive with Anterior instability (SOURCE-26+15). Near end-range the unstable shoulder may relocate, indicating at least a partial Subluxation of the Head of Humerus . Two or more directions of instability would indicate multidirectional instability which should then be followed up by a screening of unrelated joints to screen for systemic laxity:

Orthopaedic Tests

The following Shoulder - Special Tests can be used to evaluate the presence and grade of instability in various directions:Anterior:

Posterior:

Inferior:

Multidirectional:

Muscle Test

The following Muscles may be weak or inhibited with instability:

Nerve

The following tests can be performed as a cluster for a quick-screening of the related Nerve s, even if the patient is supported in a sling (SOURCE-7):

Imaging

While the diagnosis of Glenohumeral Dislocation is predominately based on clinical findings, imaging may be used to evaluate concomitant or contributing pathology:

Magnetic Resonance Imaging (MRI)- with the capacity for Bone and Soft-Tissue to be implicated in instability, MRIs may be preferable due to their ability to produce clear images of both. High sensitivity for the detection of edema also allows MRIs to recognise pathologies in their early phase (SOURCE-27). When compared to 1.5-T MRIs, the stronger 3-T MRIs offer enhanced accuracy for visualizing subtle soft tissue and bony lesions, which can be crucial for differentiating structural and dynamic pathologies related to shoulder instability and associated conditions like impingement (SOURCE-28).

  • MR-Arthrography - the complement of contrast injections distends the joint and sequesters into tears for better visualisation. For acute injury this may not be necessary as joint effusion may play a similar revealing role (SOURCE-29). MR-Arthrography is considered the modaility of choice for imaging of the Glenoid Labrum and Glenohumeral Capsular Ligaments . Injury to the Capsulo-Labro-Ligamentous Complex may be identified by the contrast fluid (or joint effusion) that distinguishes the previously attached structures (SOURCE-29). In chronic injury, displaced tissue may scar into a round mass known as theGlenoid Labrum Ovoid Mass (GLOM)which can also be visualised on MRI’s (SOURCE-29). Galolinium-based contrast material may afford a more favorable contrast-to-noise ratio and intraarticular injections may allow for better joint distension and delineation of labral lesions when compared to intravenous injections (SOURCE-29).

Radiographs (X-Rays) - are highly accessible and provide clear images of osseous structures. X-Rays may be relevant for the diagnosis of Glenohumeral Dislocation or the evaluation of secondary bony lesions of the Humerus or Glenoid Fossa, including Hill-Sachs Lesion s, Bankart Lesions or other Avulsion Fractures (SOURCE-29). In the acute setting, the following views may be relevant (SOURCE-29+13+15):

  • Anteroposterior (Grashey) View

  • Transscapular (Scapular-Y) View

  • Garth View (X-ray beam orientated 45º caudally from AP view) - demonstates anteroinferior margins of Glenoid and posterosuperior aspect of Humeral Head without requiring abduction

  • West Point View - recommended for suspected Bankart Lesion

  • Stryker Notch View - recommneded for suspected Hill-Sachs Lesion

In certain instances the addition of dynamic stress radiographs may provide better insight into the instability of the shoulder (SOURCE-29).

Computed Tomography (CT) Scan - while generally used as an alternative to MRI’s when they are contraindicated, they appear to boast similar accuracy for Glenoid Labrum assessment (SOURCE-29). The detail of CT scans allows for the evaluation of osseous structures to a standard where bone loss may be quantified (eg. glenoid index) and congential bone alterations are recognised (SOURCE-29). Glenoid Dysplasia, for example, is often visualised as a blunting and convexity of the posterior Glenoid Rim in the axial plane - a.k.a. “Lazy-J-Sign” (SOURCE-29). For CT-Arthrography, single contrast methods may be prefered at a dilution radiologist dependent (SOURCE-29). A dilution of 10ml of Ionic contrast material (320 mg of iodine per milliliter), diluted with 5 mL of local anesthetic has been described (SOURCE-29).


Treatment

Typically surgical intervention is reserved for those with recurrent episodes of dislocations; however, the patients activity levels may indicate surgery even following a single episode. For example, adolescents participating in contact sports may require surgery to prevent otherwise likely recurrence (SOURCE-9). In these cases conservative management should still first be attempted which usually involves immobilisation of 4-6 weeks before commencing a physical therapy program (SOURCE-9). Pain -free Range of Motion is carefully progressed and then followed by a progressive Strength program. Only once full Pain -free range and Strength is obtained should the athlete consider returning to sport.

Immobilisation

While immobilisation following dislocation remains standard practice it is not without criticism. Some have argued no significant reduction in recurrence beyond a week of immobilisation (SOURCE-9). While a combined position of GH Joint - Abduction and GH Joint - Internal Rotation is also considered standard, others have argued GH Joint - External Rotation produces lower recurrence rates (SOURCE-9). Ultimately the benefit of minimising the risk of further damage in the acute phase of injury must be evaluated against the detriment of immobilisation which includes muscle atrophy and restriction.

Strengthening

Conservative managment that emphasises the Strength ening of the following Muscles has displayed good to excellent outcomes in the treatment of traumatic and atraumatic instability (SOURCE-30+13):

  • Rotator Cuff - general weakness associated with anterior instability and recurrence (SOURCE-14). Additionally, smaller cross-sectional areas of the posterior rotators ( Infraspinatus and Teres Minor ) are related to anterior instability while smaller anterior rotators ( Subscapularis ) are related to posterior instability (SOURCE-38)

  • Deltoid - unlike the compressive action of the Rotator Cuff, the Deltoids generate substantial superior shear forces to either stabilise or destabilise the Humerus depending on contextual factors such as arm position and exernal load (SOURCE-39). The Deltoids notable resistance to inferior translation of the humerus increasingly diminishes as the arm elevates (SOURCE-39)

  • Coracobrachialis - affords a similar superior shear force on the Humerus as the Deltoid and is considerd a posterior stabiliser (SOURCE-39)

  • Scapula Stabilisers

Exercise selection should always be considerate of exercise selection as to not perpetuate overactive muscles (SOURCE-30).

Initial Phase - should address the altered neuromuscular control by promoting appropriate muscle recruitment with particular focus on the Rotator Cuff and other Scapula stabilisers (SOURCE-30). Strength may be increasingly emphasised as neuromuscular function is restored. In chronic or pathologic states such as a Tendinopathy , eccentric Muscle Contractions may be relevant (SOURCE-30).

Mid-Phase - adequate muscle activation and active Range of Motion should now be achieved and exercise selection may begin to address specific deficits and demands of the patient (SOURCE-30). This may include rudimentary versions of Barbell, Dumbbell and machine exercises:

  • Scapular Pinches - Swiss Robbery Pinches w/ DB’s variation - isometric, moderate load Scapular retraction exercise

  • Seated Row - or High Row variation - moderate-to-high load isotonic Scapula retraction exercise

  • Lat Pulldown - isotonic exercise that promotes downwards rotators of Scapula

  • Face Pulls - bilateral isonotic horizontal pull exercise that emphasises GH Joint - External Rotation

  • Upright Row - weighted isotonic vertical pull exercise that emphasises Upper Trapezius , Deltoid and external rotators

  • Overhead Press - isotonic strength exercise that approaches maximum range overhead

  • DB Shoulder Press - overhead press variation that loads each side independently

Late Phase - with basic asymmetries, deficits and movement quality now addressed, the patient is ready to undertake advanced stability and strength exercises throughout complex, circumstance (or sport) specific movements (SOURCE-30). Exercise selection should emphasise relevant functional patterns or kinetic chains with increasing difficulty, through parameters such as load, speed or instability (SOURCE-30).

Closed-Reduction

A non-surgical approach to return the Head of Humerus to the Glenoid Fossa of Scapula that may be conducted in a medical setting to minimise the amount of time complications have to stem from Dislocation (SOURCE-10). While there are many techniques that may be used, the majoirty use one or more of the following: traction, leverage or Scapula manipulation (SOURCE-42). The specific technique selected is ofen determined by practitioner preference and patient comfortability (SOURCE-10+42):

  • Milch Maneuver- patient lies supine and slightly upright (shoulders higher than hips). Both hands of the pracitioner apply certain forces to the affected arm:

    • One hand grasps the patients wrist and moves the arm overhead (similar to an Apprehension Test position)

    • Other hand stabilises above The Elbow and draws the Humerus superior and laterally. An axial traction force may also be added

  • FARES Method- patient lies supine with arm in neutral rotation and 90º GH Joint - Abduction . Practitioner grasps The Wrist and applies an axial tracition to the affected arm. While sustaining this traction, the arm is oscilated up and down ~5cm (anteroposterior for patient) and simultaneously brought into greater Abduction (up to 120º) and some GH Joint - External Rotation

  • Hippocratic Method- patient lies supine with arm in slight GH Joint - Abduction and neutrally rotated. The practioner grasps the patients wrist and applies a traction force along the length of the arm. The practioners heel (or a strap) applies a counter-force at the patients axilla. Sustaining this traction, GH Joint - Adduction is then added

Surgery

As a general trend first-time acute Dislocations are managed conservatively with immobilisation through the use of a sling, afterwhich treatment techniques such as those describe above may be implemented (SOURCE-30). In a similar fashion, conservative management is recommended for multidirectional instability through Strength ening before surgical capsule tightening should be considered (SOURCE-30). Surgical intervention becomes increasingly indicated with Dislocation reccurence, particularly when associated with contact or elite sport (SOURCE-30). Broadly speaking there are several surgical techniques that may be relevant for those with Glenohumeral Instability, including:

  • Arthroscopic Surgery

  • Open Surgery

  • Shoulder Arthroplasty

  • Fracture Fixation

  • Tendon Transfer

In terms of specific procedures, theBankartandLatarjetprocedures are most commonly utilised for the treatment of Glenohumeral Instability which represent a combination of the aforementioned techniques (SOURCE-30):

  • Bankart Procedure - as the name suggests this procedure is designed to repair a Bankart Lesion that occurs following Glenohumeral Dislocation . While traditionally performed through open surgery, is now more commonly performed arthroscopically (SOURCE-33+31). In either instance, the Bankart procedure retentions the Anterior Glenohumeral Joint Capsule and repairs the avulsed Glenoid Labrum using sutures or anchor sutures (SOURCE-31). While this procedure is often used as the surgical first line of attack for instability, it is reserved for less severe instances where bone loss is minimal (SOURCE-31+32)

  • Latarjet Procedure - also known as theCoracoid Bone Block Procedure, transfers the portion of the Coracoid Process of Scapula where the Conjoint tendons (formed by fusion of Short Head of Biceps and Coracobrachialis tendons) attach to the Anterior Glenoid Rim through the use of screws (SOURCE-31). In contrast to Bankart procedure, the Latarjet requires open surgery and is therefore more invasive; however, it appears to boast superior outcomes in the settings of notable boneloss and is more effective at preventing reccurent instability symptoms over the long-term (SOURCE-31+34).

For the majority of outcome measures including rates or timing of return to sport, return to pre-injury level, Range of Motion and functionality there appears to be no significant difference between the two procedures (SOURCE-35+34). The less invasive Bankart procedure did however boast significantly higher reccurrence and reoperation rates (SOURCE-36+37+32).

Following surgery The Shoulder Girdle is typically immobilised for approximately 4 weeks, with only active-assisted exercises within a “safe-zone” permitted (SOURCE-30). Other post-surgical recommendations include (SOURCE-30):


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