Thoracic Outlet Syndrome

Thoracic Outlet Syndrome, abbreviatedTOS, describes the compression of the neurovascular structures as they exit a site through the Scalenes and above the 1st Ribs , known as thethoracic outlet. In the vast majority (>90%) of cases, compression is neurological, affecting the Brachial Plexus (SOURCE-3+7+9). TOS is a major pathology of the Thoracic Spine . TOS affects roughly 3-80/1,000 of the general population and is 3-4 times more common in women and usually occurs between the ages of 20 and 50 (SOURCE-9). Compression of the same neuromuscular bundle occurring below the Clavicle is known as Pectoralis Minor Syndrome .


Structures

The Thoracic Outlet is marked by three spaces or passageways for the neurovascular structures, each with its own set of potential complications that can narrow the space and cause compression (SOURCE-7+10+11):


Pathomechanics

The specific neurovascular structures contained within this bundle include (SOURCE-3+5+6+11):

  • Brachial Plexus - compression resulting inNeurogenic TOS. Symptoms are consistent with other neurological impingements. These symptoms include Pain anywhere between the neck, face and Occipital region down to the chest, shoulder and upper extremity. Also common is altered or absent sensation, weakness or fatigue in the arm or hands. In chronic instances muscle atrophy may develop. In adults Neurogenic TOS most often occurs following muscular trauma, whereas in children it is frequently attributed to anatomic abnormalities such as Cervical Spine or irregular First Ribs , variant Scalenes or Scoliosis (SOURCE-6)

  • Subclavian Artery and Vein - compression resulting inVenous TOS, which is also referred to asPaget-Schroetter Syndrome. Symptoms include Inflammation / swelling, Pain and discolouration while the affected limb may be described as “heavy”.Venous Collateralsmay also be visible in the Upper Limb or Chest , where venous occlusion causes blood to seek alternative pathways (SOURCE-3+5). Venous TOS is often derived from excessive activity of the Upper Limb leading to hypertrophy of the Subclavius or Costoclavicular Ligament (SOURCE-6). For adolescents activity is typically related to sports participation and symptoms are usually unilateral (SOURCE-6)

  • Axillary Vein and Artery - compression resulting inAterial TOS. Symptoms are derived from ischemia (reduced blood flow) to the Upper Limb , leading to a cascade which includes claudication, Pain , pulselessness, numbness, coldness and ulcer formation (SOURCE-3+6). Aetiology is often attributed to congenital deformities such as Cervical or anomalous First Ribs , variant Scalenes or Scoliosis (SOURCE-6)

In all three spaces, the Brachial Plexus (>90%) is the predominant structure that is compressed; however, venous (5%) or aterial (1%) obstruction may occur (SOURCE-3). A variation of additional Subclavius fibres, known as theSubclavius Posticus, has also been implicated in Proximal Thoracic Outlet Syndrome (SOURCE-2).


Pathology

The following pathologies are related to Thoracic Outlet Syndrome:

The majority of Thoracic Outlet Syndrome cases occur concomitantly with Pectoralis Minor Syndrome (SOURCE-4)

Cervical Spine Ribs - the rare variant where ribs originate from C7 is highly associated with TOS, occurring in almost 30% of cases (SOURCE-6)

Trapezius - both insufficiency and excessive tone/size of the Trapezius has been described to encroach on the Thoracic Outlet and increase pressure on the contained neurovascular structures, leading to the TOS. Upper Trapezius insufficiency may cause the shoulder to depress which can structurally imdede on the outlet (SOURCE-7). Conversely, with the attachment of the Upper fibres on the Distal Clavicle (a border of the outlet), hypertonicty and/ or hypertrophy may lead to a superoposterior pull which hypothetically narrows the Costoclavicular Space of the Thoracic Outlet (SOURCE-8)


Assessment

Observation

The following observations may be related to Thoracic Outlet Syndrome:

  • Reduced Cervical Spine Lordosis

  • Poor Grip Strength

  • Muscle Atrophy along nerve distribution - seen in chronic cases

  • Radial and Ulnar Pulse examination is integral for ruling in/ out arterial compromise. Results may be more significant in certain positions such as with the arm overhead as this can narrow the neuromuscular passage (SOURCE-2)

  • Arm size - the symptomatic arm should be compared in size to the asymptomatic arm as a disparity indicates Venous TOS (SOURCE-6)

  • Formation of Dilated Collateral Veins - local to the Chest and Deltoid region suggest chronicity of Venous TOS (SOURCE-6)

  • Pain - distribution may indicate the Nerve Root heights which are compressed (SOURCE-9):

    • Upper Plexus ( C5 , C6 , C7 ) - pain extends the majority of the neck and upper shoulder girdle

    • Lower Plexus ( C8 , T1 ) - the more common form of Thoracic Outlet Syndrome, usually refers into the shoulder and descends the inner arm

Range of Motion

Arm elevation beyond 90º has the capacity to narrow the thoracic outlet space and consequently exacerbate symptoms (SOURCE-13). This narrowing of space is described to be pronounced in those with a rounded or “poor” posture ( Forward Head Posture , Scapulothoracic Joint - Protraction and an Anterior/ Inferior Shoulder) which is often associated with Thoracic Outlet Syndrome (SOURCE-13).

Orthopaedic Tests

The following Special Tests may be relevant in the diagnosis of TOS:

Palpation

Assessment of relative tone of the Scalenes and Pectoralis Minor may be relevant as hypertonicity is known to cause compression (SOURCE-4+6). Increased pressure on these muscles may be provocative. This may also help decipher whether symptoms are of PMS or Thoracic Outlet Syndrome origin.

Imaging

Radiographs (X-Rays) should reveal anomalous or asymmetric Ribs (SOURCE-6). Duplex Ultrasound (B-Mode imaging with colour Doppler) can assist in the diagnosis of Venous and Atrial TOS as it clearly identifies thrombosis of the Axillosubclavian vein (SOURCE-6).


Treatment

Treatment should always be specific to the aetiology, with conservative treatments typically more relevant in the absence of congenital deformities (SOURCE-6). Exercise therapy has been found to be an effective treatment for 50-90% of those with Thoracic Outlet Syndrome (SOURCE-13). Appropriate positioning and motion of the Scapula is a key point of focus for Thoracic Outlet Syndrome treatment (SOURCE-13).

Stretching

Stretching techniques have be advocated for Thoracic Outlet Syndrome, in particular of the Scalenes and Pectorals (SOURCE-13):

Strengthening

Strength training should be focused around posture correction, addressing asymmetries where present (SOURCE-6). Scapula positioning/ movement is a key point of focus regarding exercise selection, with emphasis on alignment of the head and Pelvis (SOURCE-13).

Initial Phase - rehabilitation should begin by promoting muscular endurance of the Scapula stabilisers, in particular the Middle and Lower Trapezius , Rhomboids and eventually the Serratus Anterior (SOURCE-13). Range should initially be restricted between 0-30º GH Joint - Flexion while in 40º GH Joint - Abduction and horizontal adduction should be minimised to avoid perpetuating injury (SOURCE-13):

Mid-Phase:

Late Phase:

Myofascial Release

Massage of the following structures may be indicated in those with Neurogenic TOS:

Mobilisation

The following Mobilisation techniques may be relevant for the treatment of Thoracic Outlet Syndrome:

Joint Play - passive accessory movements performed without active movement

Mobilisation with Movement - mobilisations applied with active movement

Surgery

Surgical restoration of blood flow through Thrombectomy or Thrombolysis may be indicated for patients with acute thrombosis of the Brachial Artery (SOURCE-6). Surgical decompression of the following structures may also be indicated (SOURCE-6):

  • Thoracic Outlet - removal of the Anterior Scalene and resection of the First Ribs

  • Cervical Ribs

  • Infraclavicular or Transaxillar - indicated for Venous TOS to ensure adequate blood flow is restored to the vein. Transaxillary decompression is also indicated for Aterial TOS where veinous reconstruction is not needed. Where reconstruction is needed an Infraclavicular and/ or Supraclavicular approach is recommended

Medication

Anticoagulants are recommended for acute Venous TOS for a minimum of 3 months, usually in conjunction with surgical intervention (SOURCE-6). This medication appears less effective in the young and often provides less benefit when used in isolation (SOURCE-6).


References

  1. Henni S, Hersant J, Ammi M, Mortaki F-E, Picquet J, Feuilloy M and Abraham P (2019) Microvascular Response to the Roos Test Has Excellent Feasibility and Good Reliability in Patients With Suspected Thoracic Outlet Syndrome.Front. Physiol.10:136. doi: 10.3389/fphys.2019.00136

  2. Crepaz-Eger, U., Lambert, S., Hörmann, R., Knierzinger, D., Brenner, E., & Hengg, C. (2022). The anatomy and variation of the coracoid attachment of the subclavius muscle in humans.Journal of Anatomy,240(2), 376–384. https://doi.org/10.1111/joa.13548

  3. Sanders, R. J., & Annest, S. J. (2014). Thoracic outlet and pectoralis minor syndromes. Seminars in Vascular Surgery, 27(2), 86–117. https://doi.org/10.1053/j.semvascsurg.2015.02.001

  4. Aktaş, İ., & Ünlü Özkan, F. (2022). Pectoralis minor syndrome.Turkish journal of physical medicine and rehabilitation,68(4), 447–455. https://doi.org/10.5606/tftrd.2023.12037

  5. Richard, H. M., 3rd, Selby, J. B., Jr, Gay, S. B., & Tegtmeyer, C. J. (1992). Normal venous anatomy and collateral pathways in upper extremity venous thrombosis.Radiographics : a review publication of the Radiological Society of North America, Inc,12(3), 527–534. https://doi.org/10.1148/radiographics.12.3.1609143

  6. Farr, S. (Ed.). (2022). Congenital and acquired deformities of the pediatric shoulder girdle. Springer.

  7. Kaplan J, Kanwal A. Thoracic Outlet Syndrome. [updated 2023 apr 10]. In: StatPearls [internet]. Treasure Island (FL): StatPearls Publishing; 2025 Jan-. Available from: https://www.ncbi.nlm.nih.gov/books/NBK557450/

  8. Philp, J., Jeong, W. J., & Paily, P. (2024). An Uncommon Culprit: Trapezius Dystonia as a Cause of Thoracic Outlet Syndrome: A Case Report. Cureus, 16(7), e63825. https://doi.org/10.7759/cureus.63825

  9. Jones, M.R., Prabhakar, A., Viswanath, O.et al.Thoracic Outlet Syndrome: A Comprehensive Review of Pathophysiology, Diagnosis, and Treatment.Pain Ther8, 5–18 (2019). https://doi.org/10.1007/s40122-019-0124-2

  10. Freischlag, J., & Orion, K. (2014). Understanding thoracic outlet syndrome.Scientifica,2014, 248163. https://doi.org/10.1155/2014/248163

  11. Standring, S. (Ed.). (2016). Gray's anatomy: The anatomical basis of clinical practice (41st ed.). Elsevier.

  12. Magee, D. J. (2014). Orthopedic physical assessment (6th ed.). Saunders.

  13. Levine, N. A., & Rigby, B. R. (2018). Thoracic Outlet Syndrome: Biomechanical and Exercise Considerations.Healthcare (Basel, Switzerland),6(2), 68. https://doi.org/10.3390/healthcare6020068

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